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Studies on Renal Disorders (Record no. 17758)

000 -LEADER
fixed length control field 06080nam a22004575i 4500
003 - CONTROL NUMBER IDENTIFIER
control field OSt
005 - DATE AND TIME OF LATEST TRANSACTION
control field 20140310150239.0
007 - PHYSICAL DESCRIPTION FIXED FIELD--GENERAL INFORMATION
fixed length control field cr nn 008mamaa
008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION
fixed length control field 101217s2011 xxu| s |||| 0|eng d
020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 9781607618577
978-1-60761-857-7
050 #4 - LIBRARY OF CONGRESS CALL NUMBER
Classification number QH573-671
082 04 - DEWEY DECIMAL CLASSIFICATION NUMBER
Classification number 571.9453
Edition number 23
264 #1 -
-- Totowa, NJ :
-- Humana Press,
-- 2011.
912 ## -
-- ZDB-2-SBL
100 1# - MAIN ENTRY--PERSONAL NAME
Personal name Miyata, Toshio.
Relator term editor.
245 10 - IMMEDIATE SOURCE OF ACQUISITION NOTE
Title Studies on Renal Disorders
Medium [electronic resource] /
Statement of responsibility, etc edited by Toshio Miyata, Kai-Uwe Eckardt, Masaomi Nangaku.
300 ## - PHYSICAL DESCRIPTION
Extent XVIII, 800p. 101 illus., 31 illus. in color.
Other physical details online resource.
440 1# - SERIES STATEMENT/ADDED ENTRY--TITLE
Title Oxidative Stress in Applied Basic Research and Clinical Practice
505 0# - FORMATTED CONTENTS NOTE
Formatted contents note Preface -- Chapter 1. Oxidative stress injury in glomerular mesangium -- Chapter 2. Transition Metals and other Forms of Oxidative Protein Damage in -- Renal Disease -- Chapter 3. Cyclo-oxygenase in the kidney and oxidative stress -- Chapter 4. Renin angiotensin system in the kidney and oxidative stress -Local Renin-Angiotensin-Aldosterone-System and NAD(P)H oxidase-dependent oxidative stress in the kidney- -- Chapter 5. Thiamine in diabetic renal disease – dietary insufficiency, renal -- washout, anti-stress gene response, therapeutic supplements, risk predictor and link to genetic susceptibility -- Chapter 6. Novel members of the globin family and their function against oxidative stress -- Clinical Aspects of Oxidative Stress in the Kidney -- Chapter 7. Hypertension -- Chapter 8. Uric acid and oxidative stress -- Chapter 9. Reactive oxygen and nitrogen species, oxidative and nitrosative -- stress and their role in the pathogenesis of acute kidney injury -- Chapter 10. Oxidative stress in the kidney: proximal tubule disorders -- Chapter 11. Iron metabolism and oxidative stress -- Chapter 12. Hypoxia, oxidative stress and the pathophysiology of contrast-media-induced nephropathy -- Chapter 13. Cardiovascular complications in renal failure: implications of advanced glycation end-products and their receptor, RAGE -- Chapter 14. Infection and the kidney -- Chapter 15. Oxidative/carbonyl stress in the renal circulation and cardiovascular renal injury -- -- Current Therapy Targeting Oxidative Stress -- Chapter 16. Renin angiotensin system -- Chapter 17. Oxidative stress in kidney injury-peroxisome proliferator-activated receptor- agonists are in control -- Chapter 18. Statin -- Chapter 19. N-acetylcysteine in kidney disease -- Chapter 20. Advanced glycation end products (AGEs) inhibitor -- Section 2. ‘Hypoxia’ -- Hypoxia Biology -- Chapter 21. Involvement of hypoxia-inducible factor 1 in physiological and pathological responses to continuous and intermittent hypoxia: Role of reactive oxygen species -- Chapter 22. Regulation of oxygen homeostasis by PHDs -- Chapter 23. Oxygen-dependent regulation of erythropoiesis -- Chapter 24. Intricate link between hypoxia and oxidative stress in chronic kidney disease -- Chapter 25. RNA interference and the regulation of renal gene expression in -- hypoxia -- Hypoxia Pathology in Renal Disorders -- Chapter 26.Cardio-renal connection: The role of hypoxia and oxidative stress -- Chapter 27. HIF in acute kidney injury – from pathophysiology to a novel approach of organ protection -- Chapter 28. Hypoxia in chronic kidney disease: the final common pathway to end stage renal disease -- Chapter 29. Oxidative stress and hypoxia in the pathogenesis of diabetic -- nephropathy -- Chapter 30. Estimation of kidney oxygenation by BOLD-MRI -- Chapter 31. Anemia and progression of chronic kidney disease -- Section 3. Novel therapeutic approaches against oxidative stress and hypoxia -- Chapter 32. Novel therapeutic approaches against hypoxia and oxidative stress,targeting intracellular sensor molecules for oxygen and oxidative stress -- Chapter 33. Endoplasmic reticulum (ER) stress as a target of therapy against oxidative stress and hypoxia -- Chapter 34. Stem cell therapy against oxidative stress and hypoxia.
520 ## - SUMMARY, ETC.
Summary, etc Conditions such as oxidative stress and hypoxia, which have a generalized impact on the oxygen metabolism, have been implicated in the genesis of kidney disease. This means that deepening our understanding of the pathobiology of oxygen metabolism in such diseases could be a fruitful path towards tangible clinical benefits. Studies in Renal Disorder collects reviews from leading researchers and clinical scientists working in exactly this field, providing an overview of the latest advances. The causal role of impaired oxygen metabolism in kidney disease has numerous clinical implications. It affects our understanding of the therapeutic benefits accruing from anti-hypertensive agents; the way we control hyperglycemia/hyperinsulinemia and hyperlipidemia; and our use of dietary approaches to the correction of obesity. The defensive mechanisms against oxidative stress, such as the Nrf2-Keap1 system, and hypoxia, such as the PHD-HIF system, have recently been explored in various cells, including kidney cells. These mechanisms include intracellular sensors for oxidative stress and hypoxia. This means that novel approaches targeting these sensors may offer clinical benefits in kidney disease in which oxidative stress and/or hypoxia is a final, common pathway.
650 #0 - SUBJECT ADDED ENTRY--TOPICAL TERM
Topical term or geographic name as entry element Life sciences.
Topical term or geographic name as entry element Nephrology.
Topical term or geographic name as entry element Cytology.
Topical term or geographic name as entry element Life Sciences.
Topical term or geographic name as entry element Oxidative Stress.
Topical term or geographic name as entry element Cell Biology.
Topical term or geographic name as entry element Nephrology.
700 1# - ADDED ENTRY--PERSONAL NAME
Personal name Eckardt, Kai-Uwe.
Relator term editor.
Personal name Nangaku, Masaomi.
Relator term editor.
710 2# - ADDED ENTRY--CORPORATE NAME
Corporate name or jurisdiction name as entry element SpringerLink (Online service)
773 0# - HOST ITEM ENTRY
Title Springer eBooks
776 08 - ADDITIONAL PHYSICAL FORM ENTRY
Display text Printed edition:
International Standard Book Number 9781607618560
856 40 - ELECTRONIC LOCATION AND ACCESS
Uniform Resource Identifier http://dx.doi.org/10.1007/978-1-60761-857-7
942 ## - ADDED ENTRY ELEMENTS (KOHA)
Source of classification or shelving scheme
Item type E-Book
Copies
Price effective from Permanent location Date last seen Not for loan Date acquired Source of classification or shelving scheme Koha item type Damaged status Lost status Withdrawn status Current location Full call number
2014-04-05AUM Main Library2014-04-05 2014-04-05 E-Book   AUM Main Library571.9453
Languages: 
English |
العربية