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Oxidative Stress in Cancer Biology and Therapy (Record no. 17783)

000 -LEADER
fixed length control field 04654nam a22004695i 4500
003 - CONTROL NUMBER IDENTIFIER
control field OSt
005 - DATE AND TIME OF LATEST TRANSACTION
control field 20140310150239.0
007 - PHYSICAL DESCRIPTION FIXED FIELD--GENERAL INFORMATION
fixed length control field cr nn 008mamaa
008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION
fixed length control field 111124s2012 xxu| s |||| 0|eng d
020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 9781617793974
978-1-61779-397-4
050 #4 - LIBRARY OF CONGRESS CALL NUMBER
Classification number QH573-671
082 04 - DEWEY DECIMAL CLASSIFICATION NUMBER
Classification number 571.9453
Edition number 23
264 #1 -
-- Totowa, NJ :
-- Humana Press :
-- Imprint: Humana Press,
-- 2012.
912 ## -
-- ZDB-2-SBL
100 1# - MAIN ENTRY--PERSONAL NAME
Personal name Spitz, Douglas R.
Relator term editor.
245 10 - IMMEDIATE SOURCE OF ACQUISITION NOTE
Title Oxidative Stress in Cancer Biology and Therapy
Medium [electronic resource] /
Statement of responsibility, etc edited by Douglas R. Spitz, Kenneth J. Dornfeld, Koyamangalath Krishnan, David Gius.
300 ## - PHYSICAL DESCRIPTION
Extent XVIII, 462 p.
Other physical details online resource.
440 1# - SERIES STATEMENT/ADDED ENTRY--TITLE
Title Oxidative Stress in Applied Basic Research and Clinical Practice
505 0# - FORMATTED CONTENTS NOTE
Formatted contents note Mitochondria-Mediated Oxidative Stress and Cancer Therapy -- The Role of Akt Pathway Signaling in Glucose Metabolism and Metabolic Oxidative Stress -- Enhancement of Cancer Therapy Using Ketogenic Diets -- Superoxide Dismutase and Cancer Therapy -- Radiosensitization and Chemosesitization of Multicellular Spheroids by 2-deoxy-D-glucose is Stimulated by a Combination of TNFα and Glucodeprivation-Induced Oxidative Stress -- Chemotherapy-Induced Oxidative Stress in Non-Targeted Normal Tissues -- Tetrahydrobiopterin and Endothelial Nitric Oxide Synthase: Implications for Radiation-Induced Endothelial Dysfunction and Normal Tissue Radiation Injury -- Amifostine and the Endogenous Cellular Antioxidant Enzyme Manganese Superoxide Dismutase in Radioprotection -- Redox Regulation of Stem Cell Compartments: The Convergence of Radiation-Induced Normal Tissue Damage and Oxidative Stress -- Functional Imaging in the Assessment of Normal Tissue Injury Following Radiotherapy -- Histone Deacetylase Inhibitors, Oxidative Stress, and Multiple Myeloma Therapy -- Curcumin, Oxidative Stress, and Cancer Therapy -- Oxidative Stress and Pancreatic Cancer -- Oxidative Stress and Photodynamic Therapy for Prostate Cancer -- Oxidative Stress in Prostate Cancer -- Role of Vitamin E in Prostate Cancer -- Pentoxifylline, Vitamin E, and Modification of Radiation Induced Fibrosis -- Antioxidants, Anorexia/Cachexia, and Oxidative Stress in Patients with Advanced-Stage Cancer -- Radiation Protection by MnSOD-Plasmid Liposome Gene Therapy -- Antioxidants and Inhibition of Cisplatin-Induced Kidney Injury: Role of Mitochondria -- Carrier-Mediated and Targeted Cancer Drug Delivery.
520 ## - SUMMARY, ETC.
Summary, etc During the last 30 years it has become clearly evident that oxidative stress and free radical biology play key roles in carcinogenesis, cancer progression, cancer therapy, and normal tissue damage that limits treatment efficacy during cancer therapy. These mechanistic observations have led to the realization that free radical biology and cancer biology are two integrally related fields of investigation that can greatly benefit from cross fertilization of theoretical constructs.   The current volume of scientific papers was assembled under the heading of Oxidative Stress in Cancer Biology and Therapy in order to stimulate the discussion of how the knowledge gained in the emerging field of oxidative stress in cancer biology can be utilized to more effectively design interventions to enhance therapeutic responses while causing fewer treatment limiting complications. The chapters contained in this volume provide highly informative emerging perspectives on how that selective enhancement of oxidative stress in cancerous tissues can be used as a target for enhancing therapeutic outcomes as well as how selective inhibition of oxidative stress could spare normal tissue damage and inhibit carcinogenesis. In this regard, the book represents an outstanding resource for both basic and translational scientists as well as clinicians interested in the field of oxidative stress and cancer therapy.
650 #0 - SUBJECT ADDED ENTRY--TOPICAL TERM
Topical term or geographic name as entry element Life sciences.
Topical term or geographic name as entry element Oncology.
Topical term or geographic name as entry element Cytology.
Topical term or geographic name as entry element Life Sciences.
Topical term or geographic name as entry element Oxidative Stress.
Topical term or geographic name as entry element Cancer Research.
Topical term or geographic name as entry element Cell Biology.
700 1# - ADDED ENTRY--PERSONAL NAME
Personal name Dornfeld, Kenneth J.
Relator term editor.
Personal name Krishnan, Koyamangalath.
Relator term editor.
Personal name Gius, David.
Relator term editor.
710 2# - ADDED ENTRY--CORPORATE NAME
Corporate name or jurisdiction name as entry element SpringerLink (Online service)
773 0# - HOST ITEM ENTRY
Title Springer eBooks
776 08 - ADDITIONAL PHYSICAL FORM ENTRY
Display text Printed edition:
International Standard Book Number 9781617793967
856 40 - ELECTRONIC LOCATION AND ACCESS
Uniform Resource Identifier http://dx.doi.org/10.1007/978-1-61779-397-4
942 ## - ADDED ENTRY ELEMENTS (KOHA)
Source of classification or shelving scheme
Item type E-Book
Copies
Price effective from Permanent location Date last seen Not for loan Date acquired Source of classification or shelving scheme Koha item type Damaged status Lost status Withdrawn status Current location Full call number
2014-04-05AUM Main Library2014-04-05 2014-04-05 E-Book   AUM Main Library571.9453
Languages: 
English |
العربية