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Varicella-zoster Virus (Record no. 17935)

000 -LEADER
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003 - CONTROL NUMBER IDENTIFIER
control field OSt
005 - DATE AND TIME OF LATEST TRANSACTION
control field 20140310150241.0
007 - PHYSICAL DESCRIPTION FIXED FIELD--GENERAL INFORMATION
fixed length control field cr nn 008mamaa
008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION
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020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 9783642127281
978-3-642-12728-1
050 #4 - LIBRARY OF CONGRESS CALL NUMBER
Classification number QR355-502
082 04 - DEWEY DECIMAL CLASSIFICATION NUMBER
Classification number 616.9101
Edition number 23
264 #1 -
-- Berlin, Heidelberg :
-- Springer Berlin Heidelberg :
-- Imprint: Springer,
-- 2010.
912 ## -
-- ZDB-2-SBL
100 1# - MAIN ENTRY--PERSONAL NAME
Personal name Abendroth, Allison.
Relator term editor.
245 10 - IMMEDIATE SOURCE OF ACQUISITION NOTE
Title Varicella-zoster Virus
Medium [electronic resource] /
Statement of responsibility, etc edited by Allison Abendroth, Ann M. Arvin, Jennifer F. Moffat.
300 ## - PHYSICAL DESCRIPTION
Extent XIV, 378 p.
Other physical details online resource.
440 1# - SERIES STATEMENT/ADDED ENTRY--TITLE
Title Current Topics in Microbiology and Immunology,
International Standard Serial Number 0070-217X ;
Volume number/sequential designation 342
505 0# - FORMATTED CONTENTS NOTE
Formatted contents note The Varicella-Zoster Virus Genome -- VZV Molecular Epidemiology -- Roles of Cellular Transcription Factors in VZV Replication -- Effects of Varicella-Zoster Virus on Cell Cycle Regulatory Pathways -- Varicella-Zoster Virus Open Reading Frame 66 Protein Kinase and Its Relationship to Alphaherpesvirus US3 Kinases -- VZV ORF47 Serine Protein Kinase and Its Viral Substrates -- Overview of Varicella-Zoster Virus Glycoproteins gC, gH and gL -- Analysis of the Functions of Glycoproteins E and I and Their Promoters During VZV Replication In Vitro and in Skin and T-Cell Xenografts in the SCID Mouse Model of VZV Pathogenesis -- Varicella-Zoster Virus Glycoprotein M -- Varicella Zoster Virus Immune Evasion Strategies -- VZV Infection of Keratinocytes: Production of Cell-Free Infectious Virions In Vivo -- Varicella-Zoster Virus T Cell Tropism and the Pathogenesis of Skin Infection -- Experimental Models to Study Varicella-Zoster Virus Infection of Neurons -- Molecular Characterization of Varicella Zoster Virus in Latently Infected Human Ganglia: Physical State and Abundance of VZV DNA, Quantitation of Viral Transcripts and Detection of VZV-Specific Proteins -- Neurological Disease Produced by Varicella Zoster Virus Reactivation Without Rash -- Varicella-Zoster Virus Neurotropism in SCID Mouse–Human Dorsal Root Ganglia Xenografts -- Rodent Models of Varicella-Zoster Virus Neurotropism -- Simian Varicella Virus: Molecular Virology -- Simian Varicella Virus Pathogenesis -- Varicella-Zoster Virus Vaccine: Molecular Genetics -- VZV T Cell-Mediated Immunity -- Perspectives on Vaccines Against Varicella-Zoster Virus Infections.
520 ## - SUMMARY, ETC.
Summary, etc Varicella-zoster virus is a common human pathogen that causes varicella (chickenpox), establishes latency in sensory nerve ganglia and can reactivate many years later as herpes zoster. Although the VZV genome is the smallest of the human herpesviruses, VZV genes encode at least 70 proteins. Molecular epidemiologic approaches based on genomic sequencing have documented the global distribution of VZV in distinct clades that reflect patterns of human migration. Contemporary molecular methods are making it possible to dissect how VZV gene products support the viral life cycle, including those that are necessary for viral replication, virion assembly and egress as well as those that permit take over of the host cell by modulating cell cycle regulation, survival and intrinsic antiviral responses. Progress is also being made in understanding the events in VZV pathogenesis and the viral tropisms for keratinocytes, T cells, dendritic cells and neurons during primary infection, latency and reactivation and the innate and adaptive host responses that modulate these events. New insights about molecular virology and pathogenesis have emerged from comparative studies of VZV and simian varicella virus. VZV is the only human herpesvirus for which vaccines to prevent both primary and recurrent infection are approved and VZV vaccines have had significant public health benefits. These achievements and new directions that are unfolding are described in this review of VZV basic and clinical research
650 #0 - SUBJECT ADDED ENTRY--TOPICAL TERM
Topical term or geographic name as entry element Medicine.
Topical term or geographic name as entry element Medical virology.
Topical term or geographic name as entry element Biomedicine.
Topical term or geographic name as entry element Virology.
700 1# - ADDED ENTRY--PERSONAL NAME
Personal name Arvin, Ann M.
Relator term editor.
Personal name Moffat, Jennifer F.
Relator term editor.
710 2# - ADDED ENTRY--CORPORATE NAME
Corporate name or jurisdiction name as entry element SpringerLink (Online service)
773 0# - HOST ITEM ENTRY
Title Springer eBooks
776 08 - ADDITIONAL PHYSICAL FORM ENTRY
Display text Printed edition:
International Standard Book Number 9783642127274
856 40 - ELECTRONIC LOCATION AND ACCESS
Uniform Resource Identifier http://dx.doi.org/10.1007/978-3-642-12728-1
942 ## - ADDED ENTRY ELEMENTS (KOHA)
Source of classification or shelving scheme
Item type E-Book
Copies
Price effective from Permanent location Date last seen Not for loan Date acquired Source of classification or shelving scheme Koha item type Damaged status Lost status Withdrawn status Current location Full call number
2014-04-05AUM Main Library2014-04-05 2014-04-05 E-Book   AUM Main Library616.9101
Languages: 
English |
العربية